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Lupus, Vol. 15, No. 11, 784-790 (2006)
DOI: 10.1177/0961203306071004
© 2006 SAGE Publications

The environment and antiphospholipid syndrome

Y Levy

O Almog

A Gorshtein

Department of Medicine ‘E', Meir Medical Center; Tel-Aviv University, Israel

Y Shoenfeld

Department of Medicine ‘B' & Center for Autoimmune Diseases, Sheba Medical Center, Incumbent of the Laura Schwarz-Kipp Chair for Research of Autoimmune Diseases, Sackler Faculty of Medicine, Tel-Aviv University, Israel; Department of Medicine ‘B' & Center for Autoimmune Diseases, Sheba Medical Center, 52621 Tel-Hashomer, Israelshoenfel{at}post.tau.ac.il

The etiology of autoimmune diseases is multifactorial. The degree to which genetic and environmental factors influence susceptibility to autoimmune diseases is poorly defined. It is believed that versatile clinical presentations of autoimmune diseases stem from various combinations of the genetic and environmental factors. One of the newly diagnosed autoimmune diseases is the antiphospholipid syndrome (APS). APS is characterized by vascular thrombosis, and/or pregnancy morbidity associated with anticardiolipin (aCL), anti-ß2-glycoprotein-I (anti-ß2GPI) and lupus anticoagulant (LAC).

Key Words: anticardiolipin • antiphospholipid • drug • infection • lupus anticoagulant


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