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Lupus
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Ischemic optic neuropathy and high-level anticardiolipin antibodies in primary Sjögren's syndrome

Daniel H Rosler

LSU Medical Center, Department of Medicine, Section of Rheumatology in New Orleans

Mandi D Conway

Medical Retina Service, LSU Medical Center in New Orleans

Juan-Manuel Anaya

LSU Medical Center, Department of Medicine, Section of Rheumatology in New Orleans

Jose F Molina

LSU Medical Center, Department of Medicine, Section of Rheumatology in New Orleans

Ronald F Carr

Pathology and Oral Pathology, LSU Medical Center in New Orleans

Azzudin E Gharavi

LSU Medical Center, Department of Medicine, Section of Rheumatology in New Orleans, New Orleans, USA

Wendell A Wilson

LSU Medical Center, Department of Medicine, Section of Rheumatology in New Orleans, New Orleans, USA

A 39-year-old woman with recurrent severe anterior ischemic optic neuropathy in the setting of Sjögren's syndrome was found to have high-titer IgM anticardiolipin antibodies (aCL) that were enhanced by the phospholipid-binding glycoprotein ß2GP1. The recognition of IgM aCL-associated vasculopathy as a possible etiologic factor in the optic neuropathy in Sjögren's syndrome may be relevant in deciding appropriate therapy.

Key Words: Antiphospholipid antibodies • Sjögren's syndrome • ischemic optic neuropathy

Lupus, Vol. 4, No. 2, 155-157 (1995)
DOI: 10.1177/096120339500400215
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